טוען...
Nuclear hormone receptor antagonism with AP-1 by inhibition of the JNK pathway
The activity of c-Jun, the major component of the transcription factor AP-1, is potentiated by amino-terminal phosphorylation on serines 63 and 73 (Ser-63/73). This phosphorylation is mediated by the Jun amino-terminal kinase (JNK) and required to recruit the transcriptional coactivator CREB-binding...
שמור ב:
| הוצא לאור ב: | Genes Dev |
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| Main Authors: | , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Cold Spring Harbor Laboratory Press
1997
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC316827/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9407028/ https://ncbi.nlm.nih.govhttps://doi.org/10.1101/gad.11.24.3351 |
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