VIRUS INFECTION RAPIDLY ACTIVATES THE P58(IPK) PATHWAY, DELAYING PEAK KINASE ACTIVATION TO ENHANCE VIRAL REPLICATION

Previously we showed that the cellular protein P58(IPK) contributes to viral protein synthesis by decreasing the activity of the anti-viral protein, PKR. Here, we constructed a mathematical model to examine the P58(IPK) pathway and investigated temporal behavior of this biological system. We find th...

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Main Authors: GOODMAN, ALAN G., TANNER, BERTRAND C. W., CHANG, STEWART T., ESTEBAN, MARIANO, KATZE, MICHAEL G.
格式: Artigo
語言:Inglês
出版: 2011
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Article
在線閱讀:https://ncbi.nlm.nih.gov/pmc/articles/PMC3152592/
https://ncbi.nlm.nih.gov/pubmed/21612809
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.virol.2011.04.020
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