Formyl Peptide Receptor Ligands Promote Wound Closure in Lung Epithelial Cells

Mitochondrial antigens released from damaged cells act as “danger signals” capable of promoting innate immune cell migration and activation via formyl peptide receptors (FPRs). Lung epithelial cells are equipped to migrate and mount innate immune responses in the context of acute lung injury. The go...

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Detaylı Bibliyografya
Asıl Yazarlar: Shao, Guohong, Julian, Mark W., Bao, Shengying, McCullers, Meghan K., Lai, Ju-Ping, Knoell, Daren L., Crouser, Elliott D.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Thoracic Society 2011
Konular:
Rapid Communication
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3095929/
https://ncbi.nlm.nih.gov/pubmed/20889801
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1165/rcmb.2010-0246RC
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