MnSOD deficiency results in elevated oxidative stress and decreased mitochondrial function but does not lead to muscle atrophy during aging

Lignende værker

• Conditional knockout of Mn-SOD targeted to type IIB skeletal muscle fibers increases oxidative stress and is sufficient to alter aerobic exercise capacity
  af: Lustgarten, Michael S., et al.
  Udgivet: (2009)
• Complex I generated, mitochondrial matrix-directed superoxide is released from the mitochondria through voltage dependent anion channels
  af: Lustgarten, Michael S., et al.
  Udgivet: (2012)
• MnSOD Overexpression Reduces Fibrosis and Pro-Apoptotic Signaling in the Aging Mouse Heart
  af: Kwak, Hyo-Bum, et al.
  Udgivet: (2015)
• Dietary restriction attenuates age-associated muscle atrophy by lowering oxidative stress in mice even in complete absence of CuZnSOD
  af: Jang, Young C., et al.
  Udgivet: (2012)
• Increased superoxide in vivo accelerates age-associated muscle atrophy through mitochondrial dysfunction and neuromuscular junction degeneration
  af: Jang, Youngmok C., et al.
  Udgivet: (2010)