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Cell surface expression of an endoplasmic reticulum resident heat shock protein gp96 triggers MyD88-dependent systemic autoimmune diseases

Heat shock proteins have been implicated as endogenous activators for dendritic cells (DCs). Without tissue distress or death, these intracellular molecules are inaccessible to surface receptor(s) on DCs, possibly to avoid uncontrolled DC activation and breakdown of immunologic tolerance. We herein...

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Autori principali: Liu, Bei, Dai, Jie, Zheng, Hong, Stoilova, Diliana, Sun, Shaoli, Li, Zihai
Natura: Artigo
Lingua:Inglês
Pubblicazione: National Academy of Sciences 2003
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC307652/
https://ncbi.nlm.nih.gov/pubmed/14668429
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.2635458100
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