Nitric oxide suppression of cellular proliferation depends on cationic amino acid transporter activity in cytokine-stimulated pulmonary endothelial cells

Inducible nitric oxide (NO) synthase (iNOS) is a stress response protein upregulated in inflammatory conditions, and NO may suppress cellular proliferation. We hypothesized that preventing l-arginine (l-arg) uptake in endothelial cells would prevent lipopolysaccharide/tumor necrosis factor-α (LPS/TN...

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Detalhes bibliográficos
Main Authors: Chicione, Louis G., Stenger, Michael R., Cui, Hongmei, Calvert, Andrea, Evans, Rebecca J., English, B. Keith, Liu, Yusen, Nelin, Leif D.
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2011
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Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3075104/
https://ncbi.nlm.nih.gov/pubmed/21239536
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00029.2010
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