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Deficiency of cationic amino acid transporter-2 protects mice from hyperoxia-induced lung injury
The pathology of acute lung injury (ALI) involves inducible nitric oxide (NO) synthase (iNOS)-derived NO-induced apoptosis of pulmonary endothelial cells. In vitro, iNOS-derived NO production has been shown to depend on the uptake of l-arginine by the cationic amino acid transporters (CAT). To test...
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| Publicado no: | Am J Physiol Lung Cell Mol Physiol |
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| Main Authors: | , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Physiological Society
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6842872/ https://ncbi.nlm.nih.gov/pubmed/30628488 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00223.2018 |
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