Many Neuronal and Behavioral Impairments in Transgenic Mouse Models of Alzheimer's Disease Are Independent of Caspase Cleavage of the Amyloid Precursor Protein

Lignende værker

• Importance of the Caspase Cleavage Site in Amyloid-β Protein Precursor
  af: Bredesen, Dale E., et al.
  Udgivet: (2010)
• Fyn Kinase Modulates Synaptotoxicity, But Not Aberrant Sprouting, in Human Amyloid Precursor Protein Transgenic Mice
  af: Chin, Jeannie, et al.
  Udgivet: (2004)
• Transsynaptic progression of amyloid-β-induced neuronal dysfunction within the entorhinal-hippocampal network
  af: Harris, Julie A., et al.
  Udgivet: (2010)
• Comparison of Neurodegenerative Pathology in Transgenic Mice Overexpressing V717F β-Amyloid Precursor Protein and Alzheimer’s Disease
  af: Masliah, Eliezer, et al.
  Udgivet: (1996)
• Reelin Depletion in the Entorhinal Cortex of Human Amyloid Precursor Protein Transgenic Mice and Humans with Alzheimer's Disease
  af: Chin, Jeannie, et al.
  Udgivet: (2007)