RAD51 supports spontaneous non-homologous recombination in mammalian cells, but not the corresponding process induced by topoisomerase inhibitors

Những quyển sách tương tự

• Overexpression of Rad51 protein stimulates homologous recombination and increases resistance of mammalian cells to ionizing radiation.
  Bằng: Vispé, S, et al.
  Được phát hành: (1998)
• DNA polymerase β overexpression stimulates the Rad51-dependent homologous recombination in mammalian cells
  Bằng: Canitrot, Yvan, et al.
  Được phát hành: (2004)
• Different Roles for Nonhomologous End Joining and Homologous Recombination following Replication Arrest in Mammalian Cells
  Bằng: Lundin, Cecilia, et al.
  Được phát hành: (2002)
• The Rad51 Paralog Rad51B Promotes Homologous Recombinational Repair
  Bằng: Takata, Minoru, et al.
  Được phát hành: (2000)
• Homologous recombination, cancer and the ‘RAD51 paradox’
  Bằng: Matos-Rodrigues, Gabriel, et al.
  Được phát hành: (2021)