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Smac mimetic-derived augmentation of chemotherapeutic response in experimental pancreatic cancer
BACKGROUND: Pancreatic ductal adenocarcinoma (PDAC) is highly resistant to conventional chemotherapy, in part due to the overexpression of inhibitors of apoptosis proteins (IAPs). Smac is an endogenous IAP-antagonist, which renders synthetic Smac mimetics attractive anticancer agents. We evaluated t...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
BioMed Central
2011
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3034706/ https://ncbi.nlm.nih.gov/pubmed/21226944 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1471-2407-11-15 |
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