Failure of Thymidine Kinase-Negative Herpes Simplex Virus To Reactivate from Latency following Efficient Establishment

Thymidine kinase-negative mutants of herpes simplex virus did not reactivate from latency in mouse trigeminal ganglia, even when their latent viral loads were comparable to those that permitted reactivation by wild-type virus. Thus, reduced establishment of latency does not suffice to account for th...

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Bibliografiska uppgifter

I publikationen:	J Virol
Huvudupphov:	Chen, Shih-Heng, Pearson, Angela, Coen, Donald M., Chen, Shun-Hua
Materialtyp:	Artigo
Språk:	Inglês
Utgiven:	American Society for Microbiology (ASM) 2004
Ämnen:	Pathogenesis and Immunity
Länkar:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC303395/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/14671133/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/JVI.78.1.520-523.2004
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