Dynamic Kv4.3–CaMKII unit in heart: an intrinsic negative regulator for CaMKII activation

AIMS: Reduction of transient outward current (I(to)) and excessive activation of Ca(2+)/Calmodulin-dependent kinase II (CaMKII) are general features of ventricular myocytes in heart failure. We hypothesize that alterations of I(to) directly regulate CaMKII activation in cardiomyocytes. METHODS AND R...

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Main Authors: Keskanokwong, Thitima, Lim, Hyun Joung, Zhang, Peng, Cheng, Jun, Xu, Lin, Lai, Dongwu, Wang, Yanggan
Formato: Artigo
Idioma:Inglês
Publicado em: Oxford University Press 2011
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3031792/
https://ncbi.nlm.nih.gov/pubmed/21148163
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/eurheartj/ehq469
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