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Dynamic Kv4.3–CaMKII unit in heart: an intrinsic negative regulator for CaMKII activation
AIMS: Reduction of transient outward current (I(to)) and excessive activation of Ca(2+)/Calmodulin-dependent kinase II (CaMKII) are general features of ventricular myocytes in heart failure. We hypothesize that alterations of I(to) directly regulate CaMKII activation in cardiomyocytes. METHODS AND R...
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| 主要な著者: | , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Oxford University Press
2011
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3031792/ https://ncbi.nlm.nih.gov/pubmed/21148163 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/eurheartj/ehq469 |
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