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Short-term exercise training protects against doxorubicin-induced cardiac mitochondrial damage independent of HSP72

Doxorubicin (Dox) is an antitumor agent used in cancer treatment, but its clinical use is limited due to cardiotoxicity. Although exercise training can defend against Dox-mediated cardiac damage, the means for this cardioprotection remain unknown. To investigate the mechanism(s) responsible for exer...

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Autors principals: Kavazis, Andreas N., Smuder, Ashley J., Min, Kisuk, Tümer, Nihal, Powers, Scott K.
Format: Artigo
Idioma:Inglês
Publicat: American Physiological Society 2010
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC2993189/
https://ncbi.nlm.nih.gov/pubmed/20833957
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00585.2010
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