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Short-term exercise training protects against doxorubicin-induced cardiac mitochondrial damage independent of HSP72
Doxorubicin (Dox) is an antitumor agent used in cancer treatment, but its clinical use is limited due to cardiotoxicity. Although exercise training can defend against Dox-mediated cardiac damage, the means for this cardioprotection remain unknown. To investigate the mechanism(s) responsible for exer...
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| Auteurs principaux: | , , , , |
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| Format: | Artigo |
| Langue: | Inglês |
| Publié: |
American Physiological Society
2010
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| Sujets: | |
| Accès en ligne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2993189/ https://ncbi.nlm.nih.gov/pubmed/20833957 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00585.2010 |
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