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STAT1-activating cytokines limit Th17 responses through both T-bet-dependent and independent mechanisms
Given the association with autoimmune disease, there is great interest in defining cellular factors that limit overactive or misdirected Th17-type inflammation. Using in vivo and in vitro models, we investigated the molecular mechanisms for cytokine-mediated inhibition of Th17 responses, focusing on...
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| Main Authors: | , , |
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| Format: | Artigo |
| Language: | Inglês |
| Published: |
2010
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| Subjects: | |
| Online Access: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2988093/ https://ncbi.nlm.nih.gov/pubmed/20974984 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1001343 |
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