Urokinase Plasminogen Activator Impairs SNP and PGE(2) Cerebrovasodilation after Brain Injury through Activation of LRP and ERK MAPK

Pial artery dilation in response to prostaglandin (PG)E(2) and the nitric oxide (NO) releaser sodium nitroprus-side (SNP) are blunted after fluid percussion brain injury (FPI), whereas responses to papaverine are unchanged. Urokinase plasminogen activator (uPA) and ERK mitogen-activated protein kina...

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Detaylı Bibliyografya
Asıl Yazarlar: Armstead, William M., Christine, Amy J., Higazi, Abd Al-Roof, Cines, Douglas B.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Mary Ann Liebert, Inc. 2008
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2948470/
https://ncbi.nlm.nih.gov/pubmed/19061381
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/neu.2008.0666
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