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Urokinase Plasminogen Activator Impairs SNP and PGE(2) Cerebrovasodilation after Brain Injury through Activation of LRP and ERK MAPK
Pial artery dilation in response to prostaglandin (PG)E(2) and the nitric oxide (NO) releaser sodium nitroprus-side (SNP) are blunted after fluid percussion brain injury (FPI), whereas responses to papaverine are unchanged. Urokinase plasminogen activator (uPA) and ERK mitogen-activated protein kina...
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| Autors principals: | , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Mary Ann Liebert, Inc.
2008
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2948470/ https://ncbi.nlm.nih.gov/pubmed/19061381 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/neu.2008.0666 |
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