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Inhibition of TIR Domain Signaling by TcpC: MyD88-Dependent and Independent Effects on Escherichia coli Virulence

Toll-like receptor signaling requires functional Toll/interleukin-1 (IL-1) receptor (TIR) domains to activate innate immunity. By producing TIR homologous proteins, microbes inhibit host response induction and improve their own survival. The TIR homologous protein TcpC was recently identified as a v...

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Bibliografiset tiedot
Päätekijät: Yadav, Manisha, Zhang, Jingyao, Fischer, Hans, Huang, Wen, Lutay, Nataliya, Cirl, Christine, Lum, Josephine, Miethke, Thomas, Svanborg, Catharina
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Public Library of Science 2010
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2944809/
https://ncbi.nlm.nih.gov/pubmed/20886104
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.ppat.1001120
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