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Early autophagic response in a novel knock-in model of Huntington disease
The aggregation of mutant polyglutamine (polyQ) proteins has sparked interest in the role of protein quality-control pathways in Huntington's disease (HD) and related polyQ disorders. Employing a novel knock-in HD mouse model, we provide in vivo evidence of early, sustained alterations of autop...
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| Main Authors: | , , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Oxford University Press
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2935855/ https://ncbi.nlm.nih.gov/pubmed/20616151 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/hmg/ddq285 |
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