Mitochondrial depolarization underlies delay in permeability transition by preconditioning with isoflurane: roles of ROS and Ca(2+)

During reperfusion, the interplay between excess reactive oxygen species (ROS) production, mitochondrial Ca(2+) overload, and mitochondrial permeability transition pore (mPTP) opening, as the crucial mechanism of cardiomyocyte injury, remains intriguing. Here, we investigated whether an induction of...

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Détails bibliographiques
Auteurs principaux: Sedlic, Filip, Sepac, Ana, Pravdic, Danijel, Camara, Amadou K. S., Bienengraeber, Martin, Brzezinska, Anna K., Wakatsuki, Tetsuro, Bosnjak, Zeljko J.
Format: Artigo
Langue:Inglês
Publié: American Physiological Society 2010
Sujets:
Cellular and Mitochondrial Metabolism
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC2928640/
https://ncbi.nlm.nih.gov/pubmed/20519447
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00006.2010
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