A study of long-term potentiation in transgenic mice over-expressing mutant forms of both amyloid precursor protein and presenilin-1

Lignende værker

• Age-Related Impairment of Synaptic Transmission But Normal Long-Term Potentiation in Transgenic Mice that Overexpress the Human APP695SWE Mutant Form of Amyloid Precursor Protein
  af: Fitzjohn, Stephen M., et al.
  Udgivet: (2001)
• Age-Related Amyloid β Deposition in Transgenic Mice Overexpressing Both Alzheimer Mutant Presenilin 1 and Amyloid β Precursor Protein Swedish Mutant Is Not Associated with Global Neuronal Loss
  af: Takeuchi, Ayano, et al.
  Udgivet: (2000)
• Profiling of Differential Expression of Genes in Mice Carrying Both Mutant Presenilin 1 and Amyloid Precursor Protein Transgenes with or without Knockout of B(2) Adrenergic Receptor Gene
  af: Zhou, Yuan, et al.
  Udgivet: (2018)
• Overexpression of Mutant Amyloid-β Protein Precursor and Presenilin 1 Modulates Enteric Nervous System
  af: Puig, Kendra L., et al.
  Udgivet: (2015)
• Presenilin 1 transgene addition to amyloid precursor protein overexpressing transgenic rats increases amyloid beta 42 levels and results in loss of memory retention
  af: Agca, Cansu, et al.
  Udgivet: (2016)