Regulation of Inflammatory Pain by Inhibition of Fatty Acid Amide Hydrolase

Although cannabinoids are efficacious in laboratory animal models of inflammatory pain, their established cannabimimetic actions diminish enthusiasm for their therapeutic development. Conversely, fatty acid amide hydrolase (FAAH), the chief catabolic enzyme regulating the endogenous cannabinoid N-ar...

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Bibliografiska uppgifter
Huvudupphovsmän: Naidu, Pattipati S., Kinsey, Steven G., Guo, Tai L., Cravatt, Benjamin F., Lichtman, Aron H.
Materialtyp: Artigo
Språk:Inglês
Publicerad: The American Society for Pharmacology and Experimental Therapeutics 2010
Ämnen:
Behavioral Pharmacology
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC2912038/
https://ncbi.nlm.nih.gov/pubmed/20375198
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1124/jpet.109.164806
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