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Repression of IP-10 by Interactions between Histone Deacetylation and Hypermethylation in Idiopathic Pulmonary Fibrosis

Targeted repression of a subset of key genes involved in tissue remodeling is a cardinal feature of idiopathic pulmonary fibrosis (IPF). The mechanism is unclear but is potentially important in disease pathogenesis and therapeutic targeting. We have previously reported that defective histone acetyla...

وصف كامل

محفوظ في:
التفاصيل البيبلوغرافية
المؤلفون الرئيسيون: Coward, William R., Watts, Keira, Feghali-Bostwick, Carol A., Jenkins, Gisli, Pang, Linhua
التنسيق: Artigo
اللغة:Inglês
منشور في: American Society for Microbiology (ASM) 2010
الموضوعات:
الوصول للمادة أونلاين:https://ncbi.nlm.nih.gov/pmc/articles/PMC2876687/
https://ncbi.nlm.nih.gov/pubmed/20404089
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.01527-09
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