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The transcription-independent mitochondrial p53 program is a major contributor to nutlin-induced apoptosis in tumor cells

Strategies to induce p53 activation in tumors that retain wild-type p53 are promising for cancer therapy. Nutlin is a potent and selective pharmacological MDM2 inhibitor that competitively binds to its p53-binding pocket, thereby leading to non-genotoxic p53 stabilization and activation of growth ar...

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Detaylı Bibliyografya
Asıl Yazarlar: Vaseva, Angelina V., Marchenko, Natalia D., Moll, Ute M.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2009
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2823264/
https://ncbi.nlm.nih.gov/pubmed/19411846
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