Milder clinical hyperimmunoglobulin E syndrome phenotype is associated with partial interleukin-17 deficiency

Mutations in the signal transducer and activator of transcription 3 (STAT3) were reported to cause hyperimmunoglobulin E syndrome (HIES). The present study investigates T helper type 17 (Th17) responses triggered by the relevant stimuli Staphylococcus aureus and Candidia albicans in five ‘classical’...

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Autores principales: van de Veerdonk, F L, Marijnissen, R, Joosten, L A B, Kullberg, B J, Drenth, J P H, Netea, M G, van der Meer, J W M
Formato: Artigo
Lenguaje:Inglês
Publicado: Blackwell Science Inc 2010
Materias:
Translational Studies
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC2802694/
https://ncbi.nlm.nih.gov/pubmed/19878510
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1365-2249.2009.04043.x
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