The effects of dexamethasone and chlorpromazine on tumour necrosis factor-alpha, interleukin-1 beta, interleukin-1 receptor antagonist and interleukin-10 in human volunteers.

समान संसाधन

• Lipopolysaccharide-induced production of tumour necrosis factor and interleukin-1 is differentially regulated at the receptor level: the role of CD14-dependent and CD14-independent pathways.
  द्वारा: Netea, M G, और अन्य
  प्रकाशित: (1998)
• Immunoglobulin D enhances the release of tumor necrosis factor-alpha, and interleukin-1 beta as well as interleukin-1 receptor antagonist from human mononuclear cells.
  द्वारा: Drenth, J P, और अन्य
  प्रकाशित: (1996)
• Increased interleukin-1alpha and interleukin-1beta production by macrophages of low-density lipoprotein receptor knock-out mice stimulated with lipopolysaccharide is CD11c/CD18-receptor mediated.
  द्वारा: Netea, M G, और अन्य
  प्रकाशित: (1998)
• Selective regulation of intercellular adhesion molecule-1 expression by interleukin-18 and interleukin-12 on human monocytes
  द्वारा: Stuyt, Rogier J L, और अन्य
  प्रकाशित: (2003)
• Effect of xanthine derivates and dexamethasone on Streptococcus pneumoniae-stimulated production of tumor necrosis factor alpha, interleukin-1 beta (IL-1 beta), and IL-10 by human leukocytes.
  द्वारा: van Furth, A M, और अन्य
  प्रकाशित: (1995)