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ETO, fusion partner in t(8;21) acute myeloid leukemia, represses transcription by interaction with the human N-CoR/mSin3/HDAC1 complex

The t(8;21) translocation between two genes known as AML1 and ETO is seen in approximately 12–15% of all acute myeloid leukemia (AML) and is the second-most-frequently observed nonrandom genetic alteration associated with AML. AML1 up-regulates a number of target genes critical to normal hematopoies...

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Udgivet i:Proc Natl Acad Sci U S A
Principais autores: Wang, Jianxiang, Hoshino, Taizo, Redner, Robert L., Kajigaya, Sachiko, Liu, Johnson M.
Format: Artigo
Sprog:Inglês
Udgivet: National Academy of Sciences 1998
Fag:
Biological Sciences
Online adgang:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC27986/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9724795/
https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.95.18.10860
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