Accelerated Pathological and Clinical Nephritis in Systemic Lupus Erythematosus-Prone New Zealand Mixed 2328 Mice Doubly Deficient in TNF Receptor 1 and TNF Receptor 2 via a Th17-Associated Pathway

Lignende værker

• Paucity of Clinical Disease despite Serological Autoimmunity and Kidney Pathology in Lupus-Prone New Zealand Mixed 2328 Mice Deficient in BAFF
  af: Jacob, Chaim O., et al.
  Udgivet: (2006)
• B cell- and BAFF-dependence of IFNα-exaggerated disease in SLE-prone NZM 2328 mice
  af: Jacob, Noam, et al.
  Udgivet: (2011)
• Global T cell dysregulation in non-autoimmune-prone mice promotes rapid development of BAFF-independent, SLE-like autoimmunity
  af: Stohl, William, et al.
  Udgivet: (2008)
• Deficiency of type I IFN receptor in lupus-prone NZM 2328 mice decreases dendritic cell numbers and activation and protects from disease
  af: Agrawal, Hemant, et al.
  Udgivet: (2009)
• Development of systemic lupus erythematosus in NZM 2328 mice in the absence of any single BAFF receptor
  af: Jacob, Chaim O., et al.
  Udgivet: (2013)