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Loss of TMEM16A Causes a Defect in Epithelial Ca(2+)-dependent Chloride Transport
Molecular identification of the Ca(2+)-dependent chloride channel TMEM16A (ANO1) provided a fundamental step in understanding Ca(2+)-dependent Cl(−) secretion in epithelia. TMEM16A is an intrinsic constituent of Ca(2+)-dependent Cl(−) channels in cultured epithelia and may control salivary output, b...
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| Autori principali: | , , , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
American Society for Biochemistry and Molecular Biology
2009
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2781414/ https://ncbi.nlm.nih.gov/pubmed/19679661 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M109.012120 |
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