Deacetylases and NF-κB in Redox Regulation of Cigarette Smoke induced Lung Inflammation: Implications in Pathogenesis of COPD

Lignende værker

• Genetic Ablation of NADPH Oxidase Enhances Susceptibility to Cigarette Smoke-Induced Lung Inflammation and Emphysema in Mice
  af: Yao, Hongwei, et al.
  Udgivet: (2008)
• Disruption of p21 Attenuates Lung Inflammation Induced by Cigarette Smoke, LPS, and fMLP in Mice
  af: Yao, Hongwei, et al.
  Udgivet: (2008)
• Current perspectives on role of chromatin modifications and deacetylases in lung inflammation of COPD
  af: Rajendrasozhan, Saravanan, et al.
  Udgivet: (2009)
• VEGFR-2 inhibition augments cigarette smoke-induced oxidative stress and inflammatory responses leading to endothelial dysfunction
  af: Edirisinghe, Indika, et al.
  Udgivet: (2008)
• Targeted disruption of NF-κB1 (p50) augments cigarette smoke-induced lung inflammation and emphysema in mice: a critical role of p50 in chromatin remodeling
  af: Rajendrasozhan, Saravanan, et al.
  Udgivet: (2010)