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Deletion of TRPC3 in mice reduces Store-Operated Ca(2+) influx and the severity of acute pancreatitis
BACKGROUND AND AIMS: Receptor–stimulated Ca(2+) influx is a critical component of the Ca(2+) signal and mediates all cellular functions regulated by Ca(2+). However, excessive Ca(2+) influx is highly toxic resulting in cell death, which is the nodal point in all forms of pancreatitis. Ca(2+) influx...
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| Main Authors: | , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2009
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2757493/ https://ncbi.nlm.nih.gov/pubmed/19622358 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1053/j.gastro.2009.07.042 |
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