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Long-term prevention of Alzheimer’s disease-like behavioral deficits in PDAPP mice carrying a mutation in Asp664
The deficits of Alzheimer’s disease (AD) are believed to result, at least in part, from neurotoxicity of β-amyloid (Aβ), a set of 38–43 amino acid fragments derived from the β-amyloid precursor protein (APP). In addition, APP generates the APP-C31 and Jcasp toxic fragments intracellularly by cleavag...
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| Autors principals: | , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2008
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2757410/ https://ncbi.nlm.nih.gov/pubmed/18485495 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbr.2008.03.035 |
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