Decay-Accelerating Factor Suppresses Complement C3 Activation and Retards Atherosclerosis in Low-Density Lipoprotein Receptor-Deficient Mice

Decay-accelerating factor (DAF; CD55) is a membrane protein that regulates complement pathway activity at the level of C3. To test the hypothesis that DAF plays an essential role in limiting complement activation in the arterial wall and protecting from atherosclerosis, we crossed DAF gene targeted...

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Detalhes bibliográficos
Main Authors: Leung, Viola W.Y., Yun, Sheng, Botto, Marina, Mason, Justin C., Malik, Talat H., Song, Wenchao, Paixao-Cavalcante, Danielle, Pickering, Matthew C., Boyle, Joseph J., Haskard, Dorian O.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Investigative Pathology 2009
Assuntos:
Regular Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2751570/
https://ncbi.nlm.nih.gov/pubmed/19729477
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2353/ajpath.2009.090183
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