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Amyloid precursor protein-induced axonopathies are independent of amyloid-β peptides

Overexpression of amyloid precursor protein (APP), as well as mutations in the APP and presenilin genes, causes rare forms of Alzheimer’s disease (AD). These genetic changes have been proposed to cause AD by elevating levels of amyloid-β peptides (Aβ), which are thought to be neurotoxic. Since overe...

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التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Stokin, Gorazd B., Almenar-Queralt, Angels, Gunawardena, Shermali, Rodrigues, Elizabeth M., Falzone, Tomás, Kim, Jungsu, Lillo, Concepción, Mount, Stephanie L., Roberts, Elizabeth A., McGowan, Eileen, Williams, David S., Goldstein, Lawrence S.B.
التنسيق:	Artigo
اللغة:	Inglês
منشور في:	Oxford University Press 2008
الموضوعات:	Articles
الوصول للمادة أونلاين:	https://ncbi.nlm.nih.gov/pmc/articles/PMC2722897/ https://ncbi.nlm.nih.gov/pubmed/18694898 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/hmg/ddn240
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Amyloid precursor protein-induced axonopathies are independent of amyloid-β peptides

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