CUL4A Abrogation Augments DNA Damage Response and Protection Against Skin Carcinogenesis

It is intuitively obvious that the ability of a cell to repair DNA damage is saturable, either by limitation of enzymatic activities, the time allotted to achieve their function, or both. However, very little is known regarding the mechanisms that establish such a threshold. Here we demonstrated tha...

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Autors principals: Liu, Liren, Lee, Sharrell, Zhang, Jianxuan, Peters, Sara B., Hannah, Jeffrey, Zhang, Yue, Yin, Yan, Koff, Andrew, Ma, Liang, Zhou, Pengbo
Format: Artigo
Idioma:Inglês
Publicat: 2009
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Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC2722740/
https://ncbi.nlm.nih.gov/pubmed/19481525
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molcel.2009.04.020
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