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c-Cbl is not required for ERK1/2-dependent degradation of Bim(EL)

Bim(EL) the most abundant Bim splice variant, is subject to ERK1/2-catalysed phosphorylation, which targets it for ubiquitination and proteasome-dependent destruction. In contrast, inactivation of ERK1/2, following withdrawal of survival factors, promotes stabilization of Bim(EL). It has been propos...

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Detalhes bibliográficos
Main Authors: Wiggins, Ceri M., Band, Hamid, Cook, Simon J.
Formato: Artigo
Idioma:Inglês
Publicado em: 2007
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2707770/
https://ncbi.nlm.nih.gov/pubmed/17884340
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cellsig.2007.08.008
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