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The subendothelial extracellular matrix modulates JNK activation by flow
Atherosclerosis begins as local inflammation of artery walls at sites of disturbed flow. The c-Jun NH(2)-terminal kinase (JNK) is thought to be one of the major regulators of flow-dependent inflammatory gene expression in endothelial cells in atherosclerosis. We now show that JNK activation by both...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2009
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2702158/ https://ncbi.nlm.nih.gov/pubmed/19286608 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCRESAHA.108.186486 |
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