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Pancreatic protease activation by alcohol metabolite depends on Ca(2+) release via acid store IP(3) receptors
Toxic alcohol effects on pancreatic acinar cells, causing the often fatal human disease acute pancreatitis, are principally mediated by fatty acid ethyl esters (non-oxidative products of alcohol and fatty acids), emptying internal stores of Ca(2+). This excessive Ca(2+) liberation induces Ca(2+)-dep...
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| Autors principals: | , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
National Academy of Sciences
2009
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2696551/ https://ncbi.nlm.nih.gov/pubmed/19528657 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0904818106 |
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