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Intracellular Zn(2+) increases contribute to the progression of excitotoxic Ca(2+) increases in apical dendrites of CA1 pyramidal neurons

Sustained intracellular Ca(2+) elevation is a well-established contributor to neuronal injury following excessive activation of NMDA-type glutamate receptors. Zn(2+) can also be involved in excitotoxic degeneration, but the relative contributions of these two cations to the initiation and progressio...

詳細記述

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書誌詳細
主要な著者: Vander Jagt, T.A., Connor, J.A., Weiss, J.H., Shuttleworth, C.W.
フォーマット: Artigo
言語:Inglês
出版事項: 2008
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC2680677/
https://ncbi.nlm.nih.gov/pubmed/19135505
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuroscience.2008.11.052
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