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Intracellular Zn(2+) increases contribute to the progression of excitotoxic Ca(2+) increases in apical dendrites of CA1 pyramidal neurons
Sustained intracellular Ca(2+) elevation is a well-established contributor to neuronal injury following excessive activation of NMDA-type glutamate receptors. Zn(2+) can also be involved in excitotoxic degeneration, but the relative contributions of these two cations to the initiation and progressio...
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| Autors principals: | , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2008
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2680677/ https://ncbi.nlm.nih.gov/pubmed/19135505 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuroscience.2008.11.052 |
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