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PPARδ agonism inhibits skeletal muscle PDC activity, mitochondrial ATP production and force generation during prolonged contraction

We have recently shown that PPARδ agonism, used clinically to treat insulin resistance, increases fat oxidation and up-regulates mitochondrial PDK4 mRNA and protein expression in resting skeletal muscle. We hypothesized that PDK4 up-regulation, which inhibits pyruvate dehydrogenase complex (PDC)-dep...

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Autors principals: Constantin-Teodosiu, Dumitru, Baker, David J, Constantin, Despina, Greenhaff, Paul L
Format: Artigo
Idioma:Inglês
Publicat: Blackwell Science Inc 2009
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC2670036/
https://ncbi.nlm.nih.gov/pubmed/19001043
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2008.164210
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