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Apolipoprotein(a), through Its Strong Lysine-binding Site in KIV(10), Mediates Increased Endothelial Cell Contraction and Permeability via a Rho/Rho Kinase/MYPT1-dependent Pathway

Substantial evidence indicates that endothelial dysfunction plays a critical role in atherogenesis. We previously demonstrated that apolipoprotein(a) (apo(a); the distinguishing protein component of the atherothrombotic risk factor lipoprotein(a)) elicits rearrangement of the actin cytoskeleton in h...

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Detaylı Bibliyografya
Asıl Yazarlar: Cho, Taewoo, Jung, Yoojin, Koschinsky, Marlys L.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Society for Biochemistry and Molecular Biology 2008
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2662143/
https://ncbi.nlm.nih.gov/pubmed/18776185
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M802648200
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