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Restoration of contractility in hyperhomocysteinemia by cardiac-specific deletion of NMDA-R1
Homocysteine (HCY) activated mitochondrial matrix metalloproteinase-9 and led to cardiomyocyte dysfunction, in part, by inducing mitochondrial permeability (MPT). Treatment with MK-801 [N-methyl-d-aspartate (NMDA) receptor antagonist] ameliorated the HCY-induced decrease in myocyte contractility. Ho...
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| Main Authors: | , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Physiological Society
2009
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2660230/ https://ncbi.nlm.nih.gov/pubmed/19181966 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00750.2008 |
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