Mitochondrial matrix metalloproteinase activation decreases myocyte contractility in hyperhomocysteinemia

Cardiomyocyte N-methyl-d-aspartate receptor-1 (NMDA-R1) activation induces mitochondrial dysfunction. Matrix metalloproteinase protease (MMP) induction is a negative regulator of mitochondrial function. Elevated levels of homocysteine [hyperhomocysteinemia (HHCY)] activate latent MMPs and causes myo...

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Detalhes bibliográficos
Main Authors: Moshal, Karni S., Tipparaju, Srinivas M., Vacek, Thomas P., Kumar, Munish, Singh, Mahavir, Frank, Iluiana E., Patibandla, Phani K., Tyagi, Neetu, Rai, Jayesh, Metreveli, Naira, Rodriguez, Walter E., Tseng, Michael T., Tyagi, Suresh C.
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2008
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Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2519211/
https://ncbi.nlm.nih.gov/pubmed/18567713
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00099.2008
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