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β-Arrestin-2 Mediates Anti-apoptotic Signaling through Regulation of BAD Phosphorylation

β-Arrestins, originally discovered as terminators of G protein-coupled receptor signaling, have more recently been appreciated to also function as signal transducers in their own right, although the consequences for cellular physiology have not been well understood. Here we demonstrate that β-arrest...

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Main Authors: Ahn, Seungkirl, Kim, Jihee, Hara, Makoto R., Ren, Xiu-Rong, Lefkowitz, Robert J.
格式: Artigo
語言:Inglês
出版: American Society for Biochemistry and Molecular Biology 2009
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在線閱讀:https://ncbi.nlm.nih.gov/pmc/articles/PMC2659243/
https://ncbi.nlm.nih.gov/pubmed/19171933
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M808463200
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