Paxillin-Y118 phosphorylation contributes to the control of Src-induced anchorage-independent growth by FAK and adhesion

Lignende værker

• Paxillin-Y118 phosphorylation contributes to the control of Src-induced anchorage-independent growth by FAK and adhesion
  af: Gelman Irwin H, et al.
  Udgivet: (2009-01-01)
• Cross-Phosphorylation and Interaction between Src/FAK and MAPKAP5/PRAK in Early Focal Adhesions Controls Cell Motility
  af: Dwyer, Sheila Figel, et al.
  Udgivet: (2014)
• Individual Cas Phosphorylation Sites Are Dispensable for Processive Phosphorylation by Src and Anchorage-independent Cell Growth
  af: Patwardhan, Parag, et al.
  Udgivet: (2006)
• Modulation of FAK and Src adhesion signaling occurs independently of adhesion complex composition
  af: Horton, Edward R., et al.
  Udgivet: (2016)
• Src and FAK Kinases Cooperate to Phosphorylate Paxillin Kinase Linker, Stimulate Its Focal Adhesion Localization, and Regulate Cell Spreading and Protrusiveness
  af: Brown, Michael C., et al.
  Udgivet: (2005)