Pemphigus Vulgaris IgG Cause Loss of Desmoglein-Mediated Adhesion and Keratinocyte Dissociation Independent of Epidermal Growth Factor Receptor

Autoantibody-induced cellular signaling mechanisms contribute to the pathogenesis of autoimmune blistering skin disease pemphigus vulgaris (PV). Recently, it was proposed that epidermal growth factor receptor (EGFR) might be involved in PV signaling pathways. In this study, we investigated the role...

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Main Authors: Heupel, Wolfgang-Moritz, Engerer, Peter, Schmidt, Enno, Waschke, Jens
Formato: Artigo
Idioma:Inglês
Publicado: American Society for Investigative Pathology 2009
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Regular Articles
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC2630556/
https://ncbi.nlm.nih.gov/pubmed/19147829
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2353/ajpath.2009.080392
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