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Proper Restoration of Excitation-Contraction Coupling in the Dihydropyridine Receptor β(1)-null Zebrafish Relaxed Is an Exclusive Function of the β(1a) Subunit

The paralyzed zebrafish strain relaxed carries a null mutation for the skeletal muscle dihydropyridine receptor (DHPR) β(1a) subunit. Lack of β(1a) results in (i) reduced membrane expression of the pore forming DHPR α(1S) subunit, (ii) elimination of α(1S) charge movement, and (iii) impediment of ar...

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Detaylı Bibliyografya
Asıl Yazarlar: Schredelseker, Johann, Dayal, Anamika, Schwerte, Thorsten, Franzini-Armstrong, Clara, Grabner, Manfred
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Society for Biochemistry and Molecular Biology 2009
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2613631/
https://ncbi.nlm.nih.gov/pubmed/19008220
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M807767200
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