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Proper Restoration of Excitation-Contraction Coupling in the Dihydropyridine Receptor β(1)-null Zebrafish Relaxed Is an Exclusive Function of the β(1a) Subunit

The paralyzed zebrafish strain relaxed carries a null mutation for the skeletal muscle dihydropyridine receptor (DHPR) β(1a) subunit. Lack of β(1a) results in (i) reduced membrane expression of the pore forming DHPR α(1S) subunit, (ii) elimination of α(1S) charge movement, and (iii) impediment of ar...

Täydet tiedot

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Bibliografiset tiedot
Päätekijät: Schredelseker, Johann, Dayal, Anamika, Schwerte, Thorsten, Franzini-Armstrong, Clara, Grabner, Manfred
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Society for Biochemistry and Molecular Biology 2009
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2613631/
https://ncbi.nlm.nih.gov/pubmed/19008220
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M807767200
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