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Abnormal Expression of Synaptic Proteins and Neurotrophin-3 in the Down Syndrome Mouse Model Ts65Dn
Down Syndrome (DS) results from triplication of the whole or distal part of human chromosome 21. DS subjects suffer from deficits in learning and memory and cognitive functions in general, and, starting from early development, their brains show dendritic and spine structural alterations and cell los...
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| Main Authors: | , , , , , |
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| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
2008
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2613315/ https://ncbi.nlm.nih.gov/pubmed/18703118 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuroscience.2008.07.025 |
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