Primary murine Chlamydia trachomatis pneumonia in B-cell-deficient mice.
Mice were rendered deficient in B-cell activity by treatment with anti-mu antibody from birth. These animals were then infected intranasally with murine Chlamydia trachomatis (murine pneumonitis agent [MoPn]). They produced neither local nor systemic antibody to MoPn but had intact delayed-type hype...
Kaydedildi:
| Yayımlandı: | Infect Immun |
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| Asıl Yazarlar: | , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
American Society for Microbiology (ASM)
1987
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC260718/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/3498690/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.55.10.2387-2390.1987 |
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